ER stress

Intraluminal hydrogen peroxide induces a permeability change of the endoplasmic reticulum membrane

Electron Microscopy / Protein Folding / Endoplasmic Reticulum Stress / Liver / Permeability / Mice / Animals / Male / Glutathione / Endoplasmic Reticulum / ER stress / Hydrogen Peroxide / Ascorbic Acid / Oxidation-Reduction / Low molecular weight / Biochemistry and cell biology / Mice / Animals / Male / Glutathione / Endoplasmic Reticulum / ER stress / Hydrogen Peroxide / Ascorbic Acid / Oxidation-Reduction / Low molecular weight / Biochemistry and cell biology

Increase in endoplasmic reticulum-associated tissue transglutaminase and enzymatic activation in a cellular model of Parkinson\'s disease

Neurobiology Of Disease / Drug interactions / Enzyme Inhibitors / Transmission Electron Microscopy / Neurobiology / Cell Differentiation / Humans / Endoplasmic Reticulum / Tunicamycin / ER stress / Neurons / Human Brain / Enzyme / Clinical Sciences / Neuroblastoma / Cellular Automaton Model / Degeneration / Retinoic Acid / Tretinoin / Membrane Protein / Enzymatic Activity / Cytoplasm / Neurosciences / Transglutaminases / Cell Differentiation / Humans / Endoplasmic Reticulum / Tunicamycin / ER stress / Neurons / Human Brain / Enzyme / Clinical Sciences / Neuroblastoma / Cellular Automaton Model / Degeneration / Retinoic Acid / Tretinoin / Membrane Protein / Enzymatic Activity / Cytoplasm / Neurosciences / Transglutaminases

HMGA1a: sequence-specific RNA-binding factor causing sporadic Alzheimer\'s disease-linked exon skipping of presenilin-2 pre-mRNA

Genetics / Humans / Cell Death / Endoplasmic Reticulum / ER stress / Alternative splicing / Genes / RNA-binding proteins / Presenilin-2 / Neurodegenerative Disease / Alternative Splicing / Base Sequence / Alzheimer Disease / Binding Site / Biochemistry and cell biology / Alternative splicing / Genes / RNA-binding proteins / Presenilin-2 / Neurodegenerative Disease / Alternative Splicing / Base Sequence / Alzheimer Disease / Binding Site / Biochemistry and cell biology

ATM deficiency induces oxidative stress and endoplasmic reticulum stress in astrocytes

DNA replication / Oxidative Stress / Endoplasmic Reticulum Stress / Cerebellum / Mice / Animals / Endoplasmic Reticulum / Astrocyte / ER stress / Phosphorylation / Laboratory / Astrocytes / Superoxide Dismutase / Mitogen Activated Protein Kinase / Clinical Sciences / Degeneration / Heat Shock Protein / Cell Cycle Proteins / DNA binding proteins / Malondialdehyde / Cell Growth / Purkinje cell / Dna Synthesis / Animals / Endoplasmic Reticulum / Astrocyte / ER stress / Phosphorylation / Laboratory / Astrocytes / Superoxide Dismutase / Mitogen Activated Protein Kinase / Clinical Sciences / Degeneration / Heat Shock Protein / Cell Cycle Proteins / DNA binding proteins / Malondialdehyde / Cell Growth / Purkinje cell / Dna Synthesis

ATM deficiency induces oxidative stress and endoplasmic reticulum stress in astrocytes

DNA replication / Oxidative Stress / Endoplasmic Reticulum Stress / Cerebellum / Mice / Animals / Endoplasmic Reticulum / Astrocyte / ER stress / Phosphorylation / Laboratory / Astrocytes / Superoxide Dismutase / Mitogen Activated Protein Kinase / Clinical Sciences / Degeneration / Heat Shock Protein / Cell Cycle Proteins / DNA binding proteins / Malondialdehyde / C-DNA Synthesis / Cell Growth / Purkinje cell / Animals / Endoplasmic Reticulum / Astrocyte / ER stress / Phosphorylation / Laboratory / Astrocytes / Superoxide Dismutase / Mitogen Activated Protein Kinase / Clinical Sciences / Degeneration / Heat Shock Protein / Cell Cycle Proteins / DNA binding proteins / Malondialdehyde / C-DNA Synthesis / Cell Growth / Purkinje cell

Increase in endoplasmic reticulum-associated tissue transglutaminase and enzymatic activation in a cellular model of Parkinson\'s disease

Neurobiology Of Disease / Drug interactions / Enzyme Inhibitors / Transmission Electron Microscopy / Neurobiology / Cell Differentiation / Humans / Endoplasmic Reticulum / Tunicamycin / ER stress / Neurons / Human Brain / Enzyme / Clinical Sciences / Neuroblastoma / Cellular Automaton Model / Degeneration / Retinoic Acid / Tretinoin / Membrane Protein / Enzymatic Activity / Cytoplasm / Neurosciences / Transglutaminases / Cell Differentiation / Humans / Endoplasmic Reticulum / Tunicamycin / ER stress / Neurons / Human Brain / Enzyme / Clinical Sciences / Neuroblastoma / Cellular Automaton Model / Degeneration / Retinoic Acid / Tretinoin / Membrane Protein / Enzymatic Activity / Cytoplasm / Neurosciences / Transglutaminases

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